Background Understanding the biological basis of complex, heritable illnesses such as
schizophrenia is facilitated by sensitive and functionally specific measures of intermediate
processes. Context processing is a theoretically motivated construct associated with
executive function. Impairments in this process have been associated with dysfunction of the
prefrontal cortex. In the present study, we evaluated whether a specific deficit in context
processing could be associated with the unexpressed genetic liability to schizophrenia …