There is growing evidence implicating congenital and acquired thrombophilias in the
pathophysiological processes underlying miscarriage, intrauterine growth restriction (IUGR)
and pre-eclampsia. Pregnancy itself is notably a hypercoagulable state, at least in part, due
to the physiological changes in the coagulation and fibrinolytic systems; this has the
potential for interaction with an acquired or heritable thrombophilia to cause adverse
experiences. Recurrent fetal loss is associated with antiphospholipid antibody syndrome …